Presentation of HLA Class I‐Derived Peptides: Potential Involvement in Allorecognition and HLA‐B27‐Associated Arthritis
Identifieur interne : 000399 ( Main/Exploration ); précédent : 000398; suivant : 000400Presentation of HLA Class I‐Derived Peptides: Potential Involvement in Allorecognition and HLA‐B27‐Associated Arthritis
Auteurs : Peter Parham [États-Unis]Source :
- Immunological Reviews [ 0105-2896 ] ; 1996-12.
English descriptors
- Teeft :
- Acute infection, Alloreactive, Alloreactive response, Allotype, Allotypes, Amino acid code, Ankylosing, Ankylosing spondylitis, Arthritis, Arthritis rheum, Autologous line, Bacterial antigens, Benjamin parham, Cell surface, Certain peptides, Chronic stimulation, Class peptide, Class peptides, Clayberger, Clin, Crossreactive, Cytolytic assay, Disease association, Disease associations, Disease susceptibility, Endogenous presentation, Epitope, Etal, Hall bowness, Heavy chain, Heavy chain allotypes, Heavy chains, Helix, Human cells, Immunol, Individual peptides, Inflammatory, Inflammatory disease, Inflammatory response, Inflammatory responses, Iransgenic mice, Lopez, Lymphocyte, Major histocompatibility, Minor alloantigens, Molecular mimicry, Molecule, Monoclonal antibodies, Ofthe, Other class, Parham, Peptide, Peptide binding, Peptide binding motif, Peptide presentation, Protective subtypes, Reactive arthritis, Rheumatoid arthritis, Shimuzu demars, Spondylitis, Subtypes, Susceptibility, Synthetic peptide, Synthetic peptides, Target cells, Taurog, Taurog hammer, Tissue antigens, Transgenic, Transgenic animals, Transgenic rats.
Url:
DOI: 10.1111/j.1600-065X.1996.tb00932.x
Affiliations:
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Le document en format XML
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type="state">Californie</region></placeName></affiliation></author></analytic><monogr></monogr><series><title level="j" type="main">Immunological Reviews</title><title level="j" type="alt">IMMUNOLOGICAL REVIEWS</title><idno type="ISSN">0105-2896</idno><idno type="eISSN">1600-065X</idno><imprint><biblScope unit="vol">154</biblScope><biblScope unit="issue">1</biblScope><biblScope unit="page" from="137">137</biblScope><biblScope unit="page" to="154">154</biblScope><biblScope unit="page-count">18</biblScope><publisher>Blackwell Publishing Ltd</publisher><pubPlace>Oxford, UK</pubPlace><date type="published" when="1996-12">1996-12</date></imprint><idno type="ISSN">0105-2896</idno></series></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0105-2896</idno></seriesStmt></fileDesc><profileDesc><textClass><keywords scheme="Teeft" xml:lang="en"><term>Acute infection</term><term>Alloreactive</term><term>Alloreactive response</term><term>Allotype</term><term>Allotypes</term><term>Amino acid 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mice</term><term>Lopez</term><term>Lymphocyte</term><term>Major histocompatibility</term><term>Minor alloantigens</term><term>Molecular mimicry</term><term>Molecule</term><term>Monoclonal antibodies</term><term>Ofthe</term><term>Other class</term><term>Parham</term><term>Peptide</term><term>Peptide binding</term><term>Peptide binding motif</term><term>Peptide presentation</term><term>Protective subtypes</term><term>Reactive arthritis</term><term>Rheumatoid arthritis</term><term>Shimuzu demars</term><term>Spondylitis</term><term>Subtypes</term><term>Susceptibility</term><term>Synthetic peptide</term><term>Synthetic peptides</term><term>Target cells</term><term>Taurog</term><term>Taurog hammer</term><term>Tissue antigens</term><term>Transgenic</term><term>Transgenic animals</term><term>Transgenic rats</term></keywords></textClass></profileDesc></teiHeader></TEI><affiliations><list><country><li>États-Unis</li></country><region><li>Californie</li></region></list><tree><country 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